Main : General Hepatitis C News/Updates Archives

April 29, 2008

Hepatitis C Complications Helped by Maintenance Interferon

A four-year study confirms that low-dosage maintenance interferon therapy prevents disease progression in those with portal hypertension or cirrhosis from Hepatitis C.

Positive Findings In Treating Patients With Advanced Hepatitis C, Study Shows

www.sciencedaily.com

ScienceDaily (Apr. 25, 2008) — The hepatitis C therapy peginterferon alfa-2b, when given as low-dose maintenance therapy, can prevent disease progression in certain patients who failed previous interferon-based hepatitis C therapies and have advanced liver disease, according to findings from a large, four-year study presented April 24 at the 43rd annual meeting of the European Association for the Study of the Liver (EASL).

The study, called COPILOT (COlchicine versus Peg-Intron LOng-Term), showed that low-dose peginterferon alfa-2b was superior to colchicine in improving the disease-free survival of patients with cirrhosis and portal hypertension, especially in patients who stayed on treatment. In the study, more than 40 percent of patients had portal hypertension, a condition of high blood pressure in the major vessel going to the liver from the gastrointestinal tract and which often accompanies liver cirrhosis. However, peginterferon alfa-2b maintenance therapy was not superior to colchicine in patients overall.

"These findings make a strong case for considering low-dose peginterferon alfa-2b as a maintenance therapy in patients with cirrhosis and portal hypertension who have failed hepatitis C eradication therapy," said principal investigator Nezam Afdhal, M.D., Chief of Hepatology at Beth Israel Deaconess Medical Center (BIDMC) and Associate Professor of Medicine at Harvard Medical School. "While other interferon maintenance therapies have been studied in the past few years in previous interferon nonresponders, these findings show, for the first time, a clinical benefit in a specific population with advanced disease," he said.

Hepatitis C virus (HCV) infection is transmitted through exposure to infected blood and affects an estimated 4 million individuals in the United States. The current standard treatment, combination therapy with pegylated interferon plus ribavirin for 24 to 48 weeks, can eradicate the virus in about 50 percent of patients. Those who do not respond and have cirrhosis are at far greater risk for developing liver cancer or liver failure, so the development of treatment strategies for these nonresponders is a priority.

Conducted at approximately 40 sites in the United States, the COPILOT study compared weight-based low-dose peginterferon alfa-2b (subcutaneous injection of 0.5 mcg/kg/wk, one-third the dose used in standard HCV combination therapy) versus colchicine (0.6 mg orally, twice daily), an anti-inflammatory and antifibrotic medication, in 555 chronic hepatitis C patients with advanced liver fibrosis who previously failed interferon-based therapies. Patient baseline characteristics were well balanced between the two study arms.

Over the four years of the randomized study, investigators monitored the patients to determine how many reached a primary endpoint, defined as death, liver transplant, hepatocellular carcinoma (liver cancer), variceal bleeding, or liver failure (increase in Child-Pugh-Turcotte [CPT] by 2 points with ascites, jaundice or encephalopathy). They analyzed their findings for all 555 patients, who received at least one dose of their assigned drug, in two ways: based on all events that occurred during the entire four years of the study, regardless of whether a patient was still taking their assigned drug or not (the "intent-to-treat" or ITT analysis), and based on only the events that occurred while patients were taking their assigned drug (the "on drug" analysis).

The investigators found a primary endpoint was reached by 17.8% (51/286) of patients in the peginterferon alfa-2b group versus 20.4% (55/269) in the colchicine group in the ITT analysis, and by 12.2% (35/286) and 16.0% (43/269) patients, respectively, in the on-drug analysis (treatment differences were not statistically significant). Among patients who had portal hypertension (42.3% and 48.0% of patients in the peginterferon alfa-2b and colchicine groups, respectively), peginterferon alfa-2b therapy resulted in significantly improved event-free survival in both the ITT and on-drug analyses (Wilcoxon p = 0.041 and 0.028, respectively). Further, variceal bleeding, a specific complication of portal hypertension, was almost abolished with peginterferon alfa-2b in both the ITT (10 vs. 1 patients) and the on-drug (10 vs. 0 patients) analyses. In the ITT analysis, hepatocellular carcinoma occurred in 7.7% and 5.9% of patients in the peginterferon alfa-2b and colchicine groups, respectively, a non-significant difference.

Overall, 49% of patients discontinued their medication before the end of the four-year study, with 36% due to failure to comply and 13% due to side effects. Peginterferon alfa-2b was generally well tolerated. Among patients who discontinued due to interferon side effects (17.1%, 49/286), the most common reason (45%, 22/49 patients) was general intolerance to interferon (e.g., due to flu-like symptoms, malaise, and other common interferon side effects).

The COPILOT study was supported by Schering-Plough Corporation, manufacturer of peginterferon alfa-2b.

Adapted from materials provided by Beth Israel Deaconess Medical Center, via EurekAlert!, a service of AAAS.

Posted by Editors at 02:15 PM --- Printer-friendly version

April 18, 2008

Development of HCV Drug Candidate Ends

Due to phase II safety trial results, Wyeth Pharmaceuticals and ViroPharma Inc. agreed to discontinue developing their Hepatitis C drug HCV-796. Executives report that HCV-796's capacity to inflict liver damage was too great to maintain its candidacy.

ViroPharma, Wyeth end development of hepatitis C drug

Philadelphia Business Journal
www.bizjournals.com

Thursday, April 17, 2008

ViroPharma Inc. and Wyeth Pharmaceuticals have decided to discontinue the development of HCV-796, their new drug candidate being developed as a potential treatment for hepatitis C.

The companies said Wednesday night the decision was based on a previously announced safety issue -- an increased risk of liver damage -- that emerged during phase-II testing of the compound.

"Clearly, this is a disappointing outcome for patients suffering from this difficult disease," said Vincent Milano, president and CEO of ViroPharma (NASDAQ:VPHM) of Exton, Pa. "Significant activities were undertaken to determine a clear path forward for HCV-796; however, the risk associated with potential hepatotoxicity ultimately posed too high of a hurdle to merit further development."

ViroPharma and Wyeth Pharmaceuticals of Collegeville, Pa., a division of Madison, N.J.-based Wyeth (NYSE:WYE), said they do not expect to continue to collaborate on future development of hepatitis C treatment candidates.

Posted by Editors at 10:13 AM --- Printer-friendly version

April 17, 2008

Can Insect Bites Spread Hepatitis?

The warmer weather is here and so are those pesky flies and insects. Learn if you should be worried about being infected with viral hepatitis from insect bites.

by Nicole Cutler, L.Ac.

Regardless of how technologically advanced a culture is, the viral hepatitis epidemic seems to be enveloping the globe. Of particular concern to the healthcare community, the viruses that cause chronic hepatitis are the most troublesome. Chronic infection with Hepatitis B or Hepatitis C can progress to advanced liver disease, a fate involving cirrhosis, liver cancer or liver failure. A vehicle for transmitting some types of viruses, some people worry that an insect bite could pass on viral hepatitis. Since many cases of chronic viral hepatitis are incurable, hepatitis prevention takes center stage. However, even the most prepared have trouble preventing insect bites.

Because scientists know that Hepatitis B and Hepatitis C are primarily spread via blood- to-blood contact, all possible breaches of the skin have been suspected in viral hepatitis transmission. Occurring when blood from an infected person enters the body of a person who is not infected, Hepatitis B and Hepatitis C are mostly spread through:

· having unprotected sex with an infected person (more likely with Hepatitis B)
· sharing drugs, needles, or “works” when injecting drugs
· needle sticks or sharp exposures on the job
· from an infected mother to her baby during birth
· being tattooed with a contaminated needle (more likely with Hepatitis C)

Hepatitis B Virus (HBV) Transmission Risks
According to the Centers for Disease Control (CDC), those at highest risk for Hepatitis B infection include:

· Persons with multiple sex partners or diagnosis of a sexually transmitted disease
· Men who have sex with men
· Sex contacts of infected persons
· Injection drug users
· Household contacts of chronically infected persons
· Infants born to infected mothers
· Infants/children of immigrants from areas with high rates of Hepatitis B infection
· Healthcare and public safety workers with exposure to blood
· Hemodialysis patients

Hepatitis C Virus (HCV) Transmission Risks
According to the CDC, those at highest risk for Hepatitis C infection include:

· Injection drug users
· Recipients of clotting factors made before 1987
· Hemodialysis patients
· Recipients of blood and/or solid organs before 1992
· People with undiagnosed liver problems
· Infants born to infected mothers
· Healthcare/public safety workers – Low risk
· People having sex with multiple partners – Low risk
· People having sex with an infected steady partner – Low risk

Mosquito Transmission
Aside from the characteristic itching and swelling involved, fears of mosquito bites spreading disease are due to its contact with blood. The insect pierces the skin, siphons out a person’s blood then repeats on a different individual – all without sterilization in between victims. Although some say there is a theoretical risk of being infected with viral hepatitis from a mosquito bite, there are no known cases worldwide. If mosquito transmission of Hepatitis B or Hepatitis C were possible, many more millions of people would likely be affected. However, mosquito bites can transmit some viruses. Mosquito-born diseases include:

· Encephalitis
· Malaria
· Dengue fever
· Rift Valley fever
· Yellow fever

Bed Bug Transmission
Similar to a mosquito’s motive, bed bugs are also bloodsuckers. Bed bug bites can create considerable anxiety and localized and occasionally systemic reactions. Different from mosquitoes, bed bugs may be a suspected transmitter of viral hepatitis:

1. HBV – Hepatitis B viral DNA can be detected in bed bugs up to six weeks after they feed on infectious blood, but no transmission of Hepatitis B infection was found in a chimpanzee model.

2. HCV – Transmission of Hepatitis C is unlikely, since Hepatitis C viral RNA is not detectable in bed bugs after an infectious blood meal.

Regardless of the presence or absence of genetic material, these critters have never been proven to transmit disease. However, if concerned about the potential for getting Hepatitis B from this insect, make sure you are vaccinated. Some suggestions for detecting bed bugs include:

· Look around. Bed bugs are large enough to see.

· Look under the mattress and in the seams, in and around the bed frame and along any cracks or peeling paint in the wall or picture frames.

· Check in the cracks of any wooden furniture, particularly antiques.

· You can also spot bed bugs’ droppings, which may be tinged with blood.

Although the thought of mosquito or bed bug bites may be disconcerting, we do share this planet with them. Without invitation, these bugs break our skin and extract our blood, actions which logically cause concern. Thankfully, the risk of being infected with HBV or HCV through insect bites is just shy of impossible. So using mosquito repellant and checking for bed bugs will make you more comfortable and spare some itching, but it won’t make a difference in your susceptibility to acquiring viral hepatitis.


References:

Hwang, W. Stephen, et al., Bed Bug Infestations in an Urban Environment, Emerging Infectious Diseases, April 2005.

http://hotels.about.com, Bed Bugs, Charlyn Keating Chisholm, About.com, 2007.

www.cdc.gov, Hepatitis B: Fact Sheet, Centers for Disease Control, 2007.

www.cdc.gov, Hepatitis C: Fact Sheet, Centers for Disease Control, 2007.

www.emedicine.com, Bedbug Bites, Robert A Schwartz, MD, MPH, WebMD, 2007.

www.hepatitisc.org.uk, FAQ, Glasgow and Scotland Hepatitis C Charity, 2007.

www.whfhhc.com, Can Hepatitis B be transmitted by a mosquito or insect bite?, Centers for Disease Control, 2007.

Posted by Editors at 04:04 PM --- Printer-friendly version

Should You See a Specialist for Hepatitis C?

Even though you may be on a first name basis with your general practitioner, research from Germany proves that your chances for successfully eliminating the Hepatitis C virus are greater when treated by a hepatologist.

by Nicole Cutler, L.Ac.

Once diagnosed with Hepatitis C, a little research will eventually reveal that the current standard treatment is pegylated interferon with ribavirin. Despite the relatively low rate of success in eliminating the virus, many may mistakenly believe that it doesn’t matter who administers the treatment. However, new research demonstrates that it is well worth the effort to seek out a specialist for monitoring your therapy.

Successful Treatment
Known as sustained virological response (SVR), successful treatment for Hepatitis C is described as the inability to detect any of the virus in the blood six months after stopping therapy. Although it is currently the only viable option for Hepatitis C, estimates of those attaining SVR with pegylated interferon and ribavirin treatment vary widely.

Regardless of a study’s country of origin, large trials evaluating Hepatitis C treatment success have reported SVR rates ranging between 30 and 80 percent. However, close examination of people fighting Hepatitis C in the real world have led researchers to suspect that the type of doctor a patient seeks help from significantly impacts SVR likelihood.

German Study
In an attempt to realize the real world impact of the type of physician administering Hepatitis C treatment, researchers at the University of Dusseldorf conducted a landmark retrospective study. At their outpatient clinics, these German researchers analyzed the records of patients receiving at least one dose of interferon treatment for Hepatitis C over a span of seven years.

After analyzing over 300 people receiving Hepatitis C treatment for the first time, approximately two thirds consulted with an expert hepatologist on a regular basis while just over one third had their interferon treatment administered and supervised by a general practitioner. Even though the characteristics of infection were similar between those working with a hepatologist and those seeing a general practitioner, the outcomes of Hepatitis C treatment were significantly different between the two groups.

Right after interferon treatment, elimination of the Hepatitis C virus was more likely for those seeing a hepatologist:

· A viral load of zero was evident in 74 percent of those seeing a hepatologist.
· A viral load of zero was evident in 48 percent of those seeing a general practitioner.

At the six-month mark when SVR is measured, those seeing a hepatologist continued to have a definite advantage:

· SVR was attained by 66 percent of those seeing a hepatologist.
· SVR was attained by 34 percent of those seeing a general practitioner.

When broken down even more, those with genotypes 1 and 4 and those with advanced liver damage specifically benefited from expert care:

· For study participants infected with genotype 1 or 4, SVR was attained by 61 percent of those seeing a hepatologist.

· For study participants infected with genotype 1 or 4, SVR was attained by 27 percent of those seeing a general practitioner.

· For study participants with advanced liver damage, SVR was attained by 69 percent of those seeing a hepatologist.

· For study participants with advanced liver damage, SVR was attained by 25 percent of those seeing a general practitioner.

These results led the German authors to conclude “Patients with…genotypes 1 and 4 or with advanced liver damage benefit from HCV therapy supervision by a specialist, probably because of less frequent treatment interruptions or dose reductions.”

What Is An Expert?
A hepatologist is a physician who has obtained additional, specialized training in liver diseases. Initially, all hepatologists are trained in general internal medicine (adult medicine) or pediatrics (children’s medicine). Some pursue additional training in gastroenterology (which includes digestive disorders involving the esophagus, stomach, small and large intestines, pancreas, gallbladder and liver). However, to be a hepatologist, a fellowship focusing solely on the liver is typically mandated. Even though a hepatologist is the most qualified type of doctor to treat liver disease, there is currently no separate board certification examination in this highly specialized field.

The field of hepatology is a rapidly changing, emerging field. Even though the standard of care for Hepatitis C has been interferon-based treatment for many years, there are many factors involved in its administration. Hepatologists are up-to-date on the latest research and discoveries for treatment modifications, length of treatment, dosage adjustments, side effect management and all of the other details related to Hepatitis C treatment.

Although most people are comfortable with their general practitioner – and they may have been the physician who originally detected their infection – an expert may be more qualified to administer Hepatitis C treatment. Especially important for people who have genotypes 1 or 4, or who already have advanced liver disease, the chance of achieving SVR is much higher when an expert liver specialist manages treatment.


References:

http://depts.washington.edu, Sagir, A., et al., Therapy outcome in patients with chronic hepatitis C: role of therapy supervision by expert hepatologists, Journal of Viral Hepatitis, September 2007.

www.acponline.org, Hepatitis C, American College of Physicians, ACP Observer, April 2006.

www.annals.org, Update in Hepatology, Willis C. Maddrey, MD, Annals of Internal Medicine, February 2001.

www.hepatitiscounselor.com, Thoughts about Hepatitis C and Liver Disease, Hepatitiscounselor.com, 2007.

www.hepato-site.net, What is a hepatologist and why do I need one?, University of Cincinnati, 2007.

www.hivandhepatitis.com, Treatment Outcomes in HCV Patients Whose Therapy is Supervised by Expert Hepatologists, hivandhepatitis.com, October 2007.

Posted by Editors at 01:50 PM --- Printer-friendly version

April 07, 2008

Improving Response to HCV Treatment

Of special interest for those who have relapsed after Hepatitis C pegylated interferon therapy: Medical experts are gaining insight into why some courses of treatment are unsuccessful. By aiming to eliminate the likely causes for previous failures, more non-responders may be good candidates for re-treatment.

by Nicole Cutler, L.Ac.

In the United States, the Hepatitis C virus (HCV) is one of the leading causes of chronic liver disease. Accounting for about 15 percent of acute viral hepatitis, 60 to 70 percent of chronic hepatitis and up to 50 percent of cirrhosis, end-stage liver disease and liver cancer, HCV is a major threat to the health of our population. While it has undergone many improvements over the past decade, the current standard of therapy for HCV has a far way to go before this virus can be considered curable. Understanding why those with chronic HCV may not respond to treatment outlines the potential for successful re-treatment.

Sustained Virologic Response
The most common way to measure HCV treatment success is via the virologic response. To measure virologic response, doctors use a blood test to measure how much virus is in the blood. While Hepatitis C RNA may be undetectable immediately following treatment, this test must be repeated six months later to see if any of the virus remained and reproduced. Also referred to as viral load, the best outcome is a sustained virologic response (SVR). When the virus remains undetectable in the blood six months (or more) following HCV therapy, SVR is considered attained. So far, studies following those with HCV for two to three years after SVR have demonstrated a low relapse rate.

While the virologic response is primarily dependent on the action of the pegylated interferon, the prevention of relapse is mostly reflective of the action of ribavirin and the maintenance of its dosing. However, relapse prevention is also affected by the following variables:

· How quickly undetectable HCV RNA is attained after the start of treatment.

· How long the patient remains on treatment after achieving undetectable HCV RNA.

Broken down by the most common HCV genotypes, the following are estimates of how frequently SVR is attained with pegylated interferon and ribavirin therapy:

· Approximately 40 to 45 percent of those with chronic HCV genotype 1, the most common strain in the United States, achieve SVR.

· Approximately 75 to 80 percent of those with chronic HCV genotypes 2 and 3 achieve SVR.

Non-Responders
While those attaining SVR may be permanently free of HCV, the remaining people on pegylated interferon and ribavirin are non-responders. However, there is plenty of evidence that previous non-responders can be re-treated successfully, eventually achieving SVR. Identifying which HCV non-responders are appropriate candidates for re-treatment requires a complete understanding of the various virologic response patterns and the pitfalls associated with achieving and maintaining virologic response.

There are many reasons that a person may not achieve SVR. The individuals without any response to interferon-based therapies are poor candidates for re-treatment. These people have no significant decline in HCV RNA during treatment and are essentially immune to the effects of interferon. Aside from those without any virological response, the four most common reasons assumed responsible for continued HCV infection after treatment are missing doses, premature discontinuation of ribavirin, insufficient ribavirin dosage and infrequent viral load testing.

1. Missing a Dose – Recent studies highlight missing a dose of peg-interferon alfa and/or ribavirin as a leading contender for not achieving SVR. Missed doses can be a result of physicians instructing their patients to temporarily stop treatment because of significant treatment-related side effects, patients missing a dose by accident or intentional skipped doses in an attempt to self-treat side effects.

2. Premature Discontinuation of Ribavirin – Stopping ribavirin too soon increases the person’s chance of viral load rebound. In those who demonstrate a slow virologic response, several studies have demonstrated that relapse can be significantly reduced in those with HCV genotype 1 patients by prolonging the duration of treatment from 48 to 72 weeks.

3. Insufficient Ribavirin Dosing – Another frequently encountered reason for HCV relapse is initiating ribavirin treatment with an insufficient dosage. Three randomized trials have demonstrated that patients who initiate treatment with a higher dose of ribavirin have a lower relapse rate.

4. Infrequent Viral Load Testing – In addition, experts believe that SVR is attained by those who quickly recognize when viral load is undetectable and their current treatment regimen is adjusted swiftly. For this reason, viral load must be assessed periodically during treatment to identify this point as soon as possible.

By recognizing the patterns associated with a poor response to interferon-based therapy, physicians can better approximate why therapy failed and who might be a good candidate for re-treatment. If a non-responder attempts pegylated interferon and ribavirin treatment again, all efforts must be made to maintain adequate, sufficient dosing for the required time interval and frequently evaluate viral load. As long as there was some type of viral response to initial treatment, virologists estimate that a sizable percentage of previous non-responders are good candidates for HCV re-treatment.


References:

http://clinicaloptions.com, Understanding HCV Nonresponse and Identifying Candidates for Retreatment, Mitchell L. Shiffman, MD, Clinical Care Options LLC, 2008.

http://digestive.niddk.nih.gov, Chronic Hepatitis C: Current Disease Management, National Institute of Diabetes and Digestive and Kidney Diseases, 2008.

Posted by Editors at 02:32 PM --- Printer-friendly version

April 04, 2008

Hepatitis C Protein May Help Fight HIV

Scientists have discovered a unique property of a segment of one of Hepatitis C's proteins - inhibition of HIV. Continued research on this Hepatitis C component could lead to a new therapy capable of preventing HIV from being sexually transmitted.

Hepatitis May Be Ally Against HIV

April 1, 2008

www.sciam.com

A part of one of the proteins of the Hepatitis C virus shows anti-HIV activity in cell cultures. Cynthia Graber reports.

Podcast Transcript: The disease hepatitis C might provide a new tool in the fight against HIV/AIDS, say scientists at the Scripps Institute and in the Netherlands. The research was published March 31st online in the Proceedings of the National Academy of Sciences.

A segment of one of the proteins of the hepatitis C virus is called C5A. Ironically, this segment, or peptide, actually actually is deadly to the hepatitis virus. So scientists wondered if it could kill the HIV virus as well. They found that in cell cultures, C5A did indeed damage HIV. It also interfered with HIV’s ability to infect cells such as the immune system’s T cells. And C5A properties are in effect at low pH, which is important if any therapy based on it were to be used by women before sex.

The researchers say that C5A has a wider range of anti-viral activity than other antimicrobial peptides. Scientists hope that the Hep C peptide research will lead to the development of antiviral therapies that could help prevent the sexual transmission of HIV.

Posted by Editors at 03:47 PM --- Printer-friendly version

1-in-4 People Have Fatty Liver Disease

With a shocking 25 percent of Americans living with non-alcoholic fatty liver disease, California researchers claim that this condition is associated with a decline in cardiac and respiratory fitness levels.

1-in-4 U.S. Adults Have Fatty Liver Disease

www.redorbit.com

Tuesday, 25 March 2008, 12:00 CDT

One out of four U.S. adults suffers from non-alcoholic fatty liver disease — liver disease characterized by excessive fat in the liver, U.S. researchers said.

Non-alcoholic fatty liver disease, the most common cause of abnormal liver enzymes, is considered by many to be a manifestation of the metabolic syndrome — belly fat, elevated blood pressure, insulin resistance, high cholesterol — which result in an increased risk of coronary heart disease.

Study leader Joanne Krasnoff of the University of California San Francisco recruited 37 adult patients with a spectrum of non-alcoholic fatty liver disease severity measured by liver biopsy.

Patients had suboptimal cardiorespiratory fitness, muscle strength, body composition and physical activity participation. More than 97 percent had a body fat percentage that put them at increased risk for morbidity and mortality and less than 20 percent met recommended guidelines for physical activity.

The study, published in the April issue of Hepatology, demonstrated lower cardiorespiratory fitness in subjects with increasing non-alcoholic fatty liver disease severity.

However, the finding raised the question of a cause-or-effect phenomenon — does cardiorespiratory fitness attenuate non-alcoholic fatty liver disease, or does increasing non-alcoholic fatty liver disease severity result in a decline in cardiorespiratory fitness? the authors asked.

Posted by Editors at 02:15 PM --- Printer-friendly version

April 01, 2008

Gregg Allman, Co-Founder of Allman Brothers, Fighting Hepatitis C

Musician Gregg Allman is undergoing treatment for Hepatitis C. As one of the estimated five million Americans with this virus, Allman's fight against Hepatitis C reminds us that this disease affects people from all walks of life.

Gregg Allman has hepatitis C

Associated Press
www.news-journalonline.com
March 28, 2008

The Allman Brothers Band won't be peakin' at the Beacon for a while.

The rock band's annual spring run of concerts at New York City's Beacon Theatre has been postponed while co-founder Gregg Allman undergoes treatments for hepatitis C. The band also canceled their upcoming performances at the Wanee Festival, which they host every year in Live Oak.

"I'm getting better but I'm still tired," Allman said in a press release from Woody Graber, a public relations consultant for concert promoter Live Nation FL. "I need to be at 110% to do the shows the way we do them."

The Allman Brothers trace their roots to Daytona Beach, where Gregg and his late brother Duane first formed early incarnations of the band.

The Beacon dates, originally set for May 5-24, will be rescheduled. The Wanee Festival will go on as scheduled on April 11-12.

Posted by Editors at 09:20 AM --- Printer-friendly version

March 31, 2008

Do You Want to Participate in a Hepatitis-C Clinical Trial?

Presently, there are many clinical trials devoted to Hepatitis C. While participating in a study offers some people a chance at improved health, the decision to enroll must be carefully considered.

by Nicole Cutler, L.Ac.

A person living with Hepatitis C could have many reasons for participating in a clinical study. However, making the decision to be a subject in a trial is far from simple. Before deciding to sign up, make certain that you have all the facts. Understanding the pros and cons involved can help a person make this important decision.

What is a Clinical Trial?
A clinical trial is a research study using humans to answer specific health questions. When conducted carefully and thoroughly, clinical trials are the safest and fastest way to find effective treatments. Clinical trials for Hepatitis C could span many purposes, including:

· Finding ways to prevent the virus from proliferating
· Testing vaccines to protect someone from becoming infected
· Improving the existing treatment
· Testing a new treatment
· Investigating options for improving quality of life

Each clinical trial has its own protocol such as:

· What types of patients may enter the study
· What the schedule of tests and procedures are
· What drugs and specific dosages are administered
· The length of the study
· How the outcomes will be measured

These criteria help to reduce the amount of variation in the study without threatening the scientific integrity of the trial. Every protocol is designed to remove medical variations that might complicate analyzing the results. As a rule, each person participating in the study must agree to the rules set out by the protocol.

To see if it works as well or better, clinical trials for Hepatitis C often compare a new product or therapy to the current standard of treatment. In a blinded study, a participant is randomly assigned to one of three groups – and not informed of which group they were assigned to. In general the categories include those who:

1. Receive the product being tested
2. Receive the existing, approved therapy
3. Receive a placebo (a product such as a sugar pill that has no therapeutic action yet looks like the product being tested)

However, placebos are rarely used in Hepatitis C trials. This is because participants with serious illness are typically afforded some kind of assumed effective therapy.

Why Volunteer for a Clinical Trial?
Since clinical trials are the only way to test new ways to fight Hepatitis C, participating puts you on the cutting edge of medicine. Typically, people with Hepatitis C enroll in a study because they have already exhausted their treatment options. Either categorized as a non-responder to pegylated interferon therapy or unable to tolerate the current therapy’s side effects, clinical trials may provide additional hope.

It is important to realize that not everyone who applies for a clinical trial will be accepted. Volunteers could be excluded based on:

· the eligibility criteria
· the number of participants needed
· any number of additional complicating factors

If interested in a clinical trial, you should learn as much as possible about it. In addition to understanding what happens during the trial, the type of health care received and any costs involved, it is important to feel comfortable discussing your concerns with the facilitators of the trial.

Pro and Con Balancing
By weighing the potential benefits against the possible risks, the decision to volunteer in a study can be clarified. Benefits to being a trial participant may include:

· being actively involved in your own health care
· gaining access to potentially new research treatments
· receiving expert medical care since the investigators are likely to be Hepatitis C specialists
· helping others by contributing to medical research

It is also important to consider both the known and unknown risks. The hazards of participating in a clinical trial may include receiving ineffective treatment, suffering from serious or life-threatening side effects or even being overwhelmed by the time investment required for the study.

An additional consideration is whether or not medical care will continue after the trial’s conclusion. Some possible questions to ask include:

· If there are side effects that linger beyond the time of the trial, will the trial’s healthcare team offer you any support?

· If the treatment was beneficial, will it be continued after the trial is over?

Finding a Hepatitis C Trial
Clinical trials can be sponsored by an organization such as a pharmaceutical company, a federal agency (such as the National Institutes of Health or Veterans Administration) or an individual (such as a physician or health care provider). Determined by the sponsor, trial locations generally are at universities, medical centers, clinics, doctor’s offices, hospitals and other research sites.

You can find information about clinical trials currently being conducted by searching www.clinicaltrials.gov. This website is updated regularly and offers information on each trial’s purpose, who is eligible to participate, locations and phone numbers to call for more information. As of late March 2008, a search on this website revealed 184 interventional trials currently recruiting or about to recruit volunteers for a Hepatitis C study. Once on this site, there is an option to refine your search using various parameters, such as location of study, participant age range, conditions included and the intervention being studied. Since there is often a long list of eligibility criteria, make certain that you are a match before proceeding with an inquiry.

Participating in a clinical study is not for everyone. Taking part in an as yet, unproven therapy is frightening for some and exciting for others. However, being thorough in your evaluation of any clinical trial will help you make this potentially life-changing decision. Especially for those who feel as if they’ve run out of Hepatitis C treatment options, enrolling in a clinical trial can offer the right person another chance at returning to health.


References:

http://clinicalcenter.nih.gov, Are Clinical Studies for You?, National Institutes of Health, 2008.

www.clinicaltrials.gov, Search for Clinical Trials, US National Institutes of Health, 2008.

www.brightsurf.com, Hospitals that participate in clinical trials may provide better patient care, Journal of the American Medical Association, March 2008.

www.fda.gov, Basic Questions and Answers about Clinical Trials, US Food and Drug Administration, 2008.

Posted by Editors at 10:28 AM --- Printer-friendly version

March 27, 2008

Researchers Find a Way to Interrupt Hepatitis C Virus

Scripps researchers found the protein needed to assemble Hepatitis C by using technology borrowed from molecular genetics. By altering the protein NS5A, this discovery may have significant impact on the development of new therapeutic agents.

New Method Disrupts Hepatitis C Virion Production

www.sciencedaily.com

ScienceDaily (Mar. 24, 2008) — HCV is a significant human pathogen, infecting more than three percent of the world's population. The incidence of infection in the United States has been estimated to be as high as 4 million cases. In the March issue of the journal PLoS Pathogens, Timothy Tellinghuisen, an assistant professor in the Department of Infectology at Scripps Florida, and his colleagues describe how they used mutations of the viral NS5A phosphoprotein to disrupt virus particle production at an early stage of assembly. NS5A has long been proposed as a regulator of events in the HCV life cycle, but exactly how it orchestrates these events has been unclear.

"The interesting thing about this mutant is that while it triggers totally normal RNA replication, it causes severe defects in the output of infectious virus--in fact, it releases no infectious virus that we can detect," says Tellinghuisen. "And though this discovery isn't a cure for HCV, it is an important research tool that stops the assembly pathway." Total disruption of the replication process would be a cure for the disease, he adds, and that's the team's long-term goal.

HCV infection is roughly five to seven times more prevalent than HIV, underscoring the pandemic nature of HCV infection. HCV occurs when blood from an infected person enters the body of someone who is not infected. Most new HCV infections are due to illegal drug injections and sharing needles. However, those who had blood transfusions prior to blood donor screening in 1991, healthcare workers who had needle stick accidents, and hemodialysis patients are also at risk for developing HCV infection.

The virus predominantly infects the liver, and following many decades of virus reproduction serious disease such as hepatitis (liver inflammation), cirrhosis (liver scarring), and carcinoma (liver cancer) develop. Ultimately, HCV infection destroys the liver, resulting in death. Attempts at curing HCV infections with drug therapy have been only marginally successful.

Before more effective therapies can be developed, scientists need to understand, at the molecular level, the detailed mechanisms HCV uses to infect cells, replicate itself, assemble progeny virus, and exit the cell. Each of these processes could potentially be a target for a new drug to eliminate HCV infection. HCV, like all viruses, requires the normal cellular machinery for its replication and has developed strategies to utilize normal cell physiology for its own benefit (often to the detriment of the host).

The Tellinghuisen team, which includes Research Assistants Katie L. Foss and Jason Treadaway, has focused recent efforts on NS5A to understand the regulation of events used by the virus to assemble infectious copies of itself and exit the cell. NS5A is a three-domain protein, which means it is comprised of three compactly folded regions roughly 50 to 300 amino acids in length. The requirement of domains I and II for RNA replication is well documented. NS5A domain III, however, is not required for RNA replication, and the function of this region in the HCV life cycle is unknown.

Using standard molecular biology, the researchers removed from domain III of NS5A a coding sequence corresponding to roughly 15 amino acids. Then they generated a clone of the virus, transcribed the RNA from that clone, and purified the RNA. This RNA, which is directly infectious, was then transfected into a liver cell line where it produced all the HCV proteins that are encoded by that RNA genome.

"Those proteins assemble in the cell to make a structure called a replicase that then copies the viral RNA," Tellinghuisen explains. "We measured that RNA accumulation and observed no defect in RNA replication, but found, surprisingly, that no infectious viral particles were released from the cells." The team also found that no viral RNA nor nucleocapsid protein are released from cells, indicating that an early event in virus assembly had been affected.

Using genetic mapping and biochemical analyses, the authors were able to show that their deletion altered a phosphorylation signal controlling the switch from RNA replication to virus particle assembly. This signal was attributed to the activity of a cellular kinase that when inhibited by genetic or chemical means led to a reduction in infectious virus production without altering HCV RNA replication.

"These data provide the first evidence for a function of domain III of NS5A and implicate NS5A as an important regulator of the RNA replication and virion assembly of HCV," Tellinghuisen says. "The ability to uncouple virus production from RNA replication may be useful in understanding HCV assembly and may become therapeutically important."

Charles M. Rice, head of the Center for the Study of Hepatitis C at Rockefeller University, comments, "This is a spectacular advance linking a specific phosphorylation event by a cellular kinase to hepatitis C virus assembly. Remarkably, the target is a viral nonstructural protein, NS5A, and the data point to a pivotal role for this protein in regulating RNA amplification and infectious virus production. These new data make this multifunctional protein an even more attractive target for developing new anti-virals for treating hepatitis C."

This project was funded by a Career Development Award from the National Institutes of Allergy and Infectious Diseases of the National Institutes of Health, and by the State of Florida.

Adapted from materials provided by Scripps Research Institute, via EurekAlert!, a service of AAAS.

Posted by Editors at 04:21 PM --- Printer-friendly version

March 20, 2008

Taribavirin Showing Lower Anemia Rates than Ribavirin

When combined with pegylated interferon for Hepatitis C treatment, taribavirin may be a better option than ribavirin. Compared to ribavirin, preliminary results of a Phase IIb trial show that taribavirin is similar in viral load reduction yet superior with fewer incidences of treatment related anemia.

Valeant Pharmaceuticals Reports Encouraging Phase IIb Results at Treatment Week 12 for Taribavirin

March 17, 2008

www.businesswire.com

ALISO VIEJO, Calif.--(BUSINESS WIRE)--Valeant Pharmaceuticals (NYSE:VRX) today reported results at the treatment week 12 analysis point for the Phase IIb clinical trial for its antiviral compound, taribavirin, a prodrug of ribavirin in development for the treatment of chronic hepatitis C in conjunction with a pegylated interferon.

The Phase IIb trial is a U.S. multi-center, randomized, parallel, open-label study in 278 treatment-naïve, genotype 1 patients evaluating taribavirin at 20 mg/kg, 25 mg/kg, and 30 mg/kg per day in combination with pegylated interferon alfa-2b. The control group is being administered weight-based dose ribavirin (800/1000/1200/1400mg daily) and pegylated interferon alfa-2b. Overall treatment duration is 48 weeks with a post-treatment follow-up period of 24 weeks. The primary endpoints for this study are viral load reduction at treatment week 12 and anemia rates throughout the study.

The 12-week early viral response (EVR) data from the Phase IIb study showed comparable reductions in viral load for weight-based doses of taribavirin and ribavirin. The anemia rate was statistically significantly lower for patients receiving taribavirin in the 20mg/kg and 25mg/kg arms versus the ribavirin control arm.

Key Efficacy and Safety Data Table at Treatment Week 12 (ITT Population)

TBV 20 mg/kg

n = 67

TBV 25 mg/kg

n = 70

TBV 30 mg/kg

n = 68

RBV 800-1400mg

n = 70

Responders(a)
43 (64.2%) 40 (57.1%) 37 (54.4%) 36 (51.4%)

Undetectable(b)
28 (41.8%) 29 (41.4%) 17 (25.0%) 22 (31.4%)

Anemia rate(c)
6 (9.0%) 5 (7.1%) 10 (14.7%) 17 (24.3%)

(a) HCV RNA undetectable (less than 100 copies per mL) or ≥2-log decrease in viral load using the NGI SuperQuant Assay
(b) HCV RNA less than 100 copies per mL

(c) Anemia rate defined as percentage of patients with Hgb level < 10 g/dL. p=0.022 for 20mg/kg and p=0.009 for 25mg/kg

The most common adverse events were fatigue, nausea, flu-like symptoms, headache and diarrhea. The incidence rates among treatment arms were generally comparable except with respect to diarrhea, where diarrhea was approximately twice as common in taribavirin patients as ribavirin patients. However, the diarrhea was generally mild and not treatment limiting for taribavirin or ribavirin patients.

In 2006 Valeant released data from its Phase III VISER1 and VISER2 trials of taribavirin, administered in a fixed dose of 600 mg BID (approximately equivalent to 13-18 mg/kg), in which taribavirin did not meet its primary efficacy endpoint of comparable efficacy to weight-based dose ribavirin. In 2007, the company began this Phase IIb study to evaluate higher doses of taribavirin than used in the VISER trials, while also employing a weight-based dosing regimen, consistent with the dosing regimen for ribavirin.

“Ribavirin continues to be a necessary part of hepatitis C treatment, and clinicians would welcome a ribavirin prodrug with a profile of similar efficacy with decreased anemia,” stated Fred Poordad, M.D., Chief of Hepatology at the Center for Liver Disease and Transplant, Cedars-Sinai Medical Center, Los Angeles, CA. “A drug with fewer required dose reductions for anemia or a reduced need for adjunctive growth factor therapy may subsequently improve treatment adherence and overall response rate.”

“We are encouraged that these data suggest that weight-based dosing with taribavirin at higher doses may have a role in the treatment of patients infected with hepatitis C while continuing to produce lower anemia than ribavirin,” said J. Michael Pearson, Valeant’s chairman and chief executive officer. “However, we caution that these results only represent data from the first 12 weeks of a 72-week Phase II trial that was designed to identify appropriate doses for further development. We will use these data to explore the best options for taribavirin’s continued role in our portfolio, including consideration of partnering options.”

Patient Demographics

20 mg/kg

n = 67
25 mg/kg

n = 70
30 mg/kg

n = 68
Ribavirin

n = 70

Age (yrs, mean) 48.5 47.5 49.6 49.7
Gender (female) 52.2% 35.7% 36.8% 31.4%
Race (Caucasian) 74.6% 58.6% 61.8% 64.3%

Weight (>75 kg)(d)
64.2% 61.4% 63.2% 62.9%

Plasma HCV RNA ≥ 2 million copies(d)
73.1% 72.9% 72.1% 70.0%

(d) Study stratified patients by weight and viral load

In the study, the following percentages of patients completed 12 weeks of treatment in the taribavirin and ribavirin arms: 20mg/kg: 87.0%; 25mg/kg: 80.0%; 30mg/kg: 82.6%; ribavirin 74.3%. The following percentages of patients had adverse events leading to dose modification or interruption of taribavirin or ribavirin: 20mg/kg: 11.9%; 25mg/kg: 15.7%; 30mg/kg: 25.0%; ribavirin 28.6%.

In the Intent to Treat Analysis, the Rapid Virological Responses (RVR - virus undetectable at treatment week 4) in this study were 20mg/kg: 16.4%; 25mg/kg: 14.3%; 30mg/kg: 16.2 %; ribavirin: 11.4%.

Conference Call and Webcast Information:

Valeant will host a conference call and webcast on Thursday, March 27, 2008 at 12:00 p.m. EDT (9:00 a.m. PDT) to discuss the Company’s Strategic Plan as well as the results from this Phase IIb clinical trial. A webcast of this event will be available live over the Internet along with a slide presentation. The webcast may be accessed through the investor relations section of Valeant’s corporate Web site at www.valeant.com. The dial-in number to participate on this call is (877) 295-5743, confirmation code 39808652. International callers should dial (706) 679-0845, confirmation code 39808652. Interested parties will have access via the Internet and on the conference call to ask questions following the presentation. A replay will be available approximately two hours following the conclusion of the conference call and can be accessed by dialing (800) 642-1687, or (706) 645-9291, confirmation code 39808652.

About Taribavirin

Taribavirin is an investigational compound that has not been found by the Food and Drug Administration (FDA) or any other regulatory agency to be safe or effective in the diagnosis, mitigation, treatment or cure of any disease or illness. It may not be sold or promoted in the United States unless and until FDA has approved a New Drug Application. Similar restrictions apply in other countries.

About Valeant

Valeant Pharmaceuticals International (NYSE:VRX) is a global specialty pharmaceutical company that develops, manufactures and markets a broad range of pharmaceutical products primarily in the areas of neurology, infectious disease and dermatology. More information about Valeant can be found at www.valeant.com.

FORWARD-LOOKING STATEMENTS

This press release contains forward-looking statements, including, but not limited to, statements regarding the potential efficacy and safety of taribavirin in the treatment of hepatitis C, and the continuing role of ribavirin or taribavirin in the treatment of hepatitis C, that are based on management’s current expectations and involve risks and uncertainties, including, but not limited to, risks and uncertainties relating to the clinical development of new products, regulatory approval processes, that results from treatment week 12 in a phase IIb clinical trial are not necessarily predictive of the entire phase IIb trial or a phase III trial, and other risks detailed from time to time in the company’s SEC filings. The company cautions the reader that these factors, as well as other factors described in its SEC filings, are among the factors that could cause actual results to differ materially from the expectations described in the forward-looking statements. The company also cautions the reader that undue reliance should not be placed on any of the forward-looking statements, which speak only as of the date of this press release. The company undertakes no responsibility to update any of these forward-looking statements to reflect events or circumstances after the date of this press release or to reflect actual outcomes.

Contacts

Valeant Pharmaceuticals
Laurie W. Little, 949-461-6002

Posted by Editors at 09:04 AM --- Printer-friendly version

March 17, 2008

3 Healthy Lifestyle Choices to Make Now

People with Hepatitis C can slow the liver's cycle of inflammation by making these three healthy lifestyle changes. By eliminating certain risk factors, one can live a long life with HCV.

by Nicole Cutler, L.Ac.

The number of people affected by Hepatitis C continues to grow. Unfortunately, the medicines used to treat this virus have not yet been able to defeat it. As of 2008, the current standard of treatment for the Hepatitis C virus (HCV), pegylated interferon and ribavirin, remains effective for approximately only half of all cases. Although pegylated interferon and ribavirin can’t help millions of people get rid of this virus, Hepatitis C doesn’t have to be a death sentence. Even though those living with chronic HCV are at a high risk of developing liver cirrhosis and/or liver cancer, eliminating three vices can prevent a worsening of liver health.

Understanding Liver Inflammation
Living with chronic HCV means constantly battling liver inflammation. If this inflammation rages unabatedly, it causes liver disease to progress. The progressive cascade of Hepatitis C and liver inflammation is as follows:

· HCV results in the death of liver cells.

· The death of liver cells triggers the dispatching of inflammatory cells to the affected area. Inflammation begins the processes that lead to fibrosis, the body’s response to liver damage.

· Inflammation triggers a reaction by a group of cells in the liver called stellate cells.

· Infected and inflamed liver cells release chemical signals (called cytokines), which activate leukocytes (white blood cells) from outside the liver to travel to the affected area.

· The cytokines and leukocytes team up with Kupffer cells to signal the stellate cells to produce and lay down collagen fibers between liver cells. A fibrous protein that forms scar tissue, collagen is the body’s attempt to limit the spread of infection to other cells.

· Normally, as an infection or injury resolves, the collagen matrix enclosing the injury is dissolved and activated stellate cells die off, allowing the tissue to return to normal. In chronic HCV, this matrix of collagen grows more rapidly than it can dissolve.

· The collagen builds up scar tissue around cells causing living liver cells to lose their access to the nutrient and oxygen rich blood flow.

· The restricted access to blood causes even more quarantined liver cells to die. As such, HCV progressively scars the liver.

This vicious cycle of inflammation causing scar tissue must be stopped to prevent a person’s chronic HCV from causing more and more liver damage.

Vice Elimination
According to Norah Terrault, MD, MPH, from the University of California, San Francisco, “Hepatitis C is a major public health concern and the number of patients developing complications of chronic disease is on the rise. It is essential that we identify risk factors that can be modified to prevent and/or lessen the progression of HCV to fibrosis, cirrhosis and even liver cancer. These complications of chronic HCV infection will significantly contribute to the overall burden of liver disease in the U.S. and will continue to increase in the next decade.”

By eliminating three unhealthful habits, people with HCV can single-handedly reduce the inflammation their liver must contend with. Although any toxin puts a greater strain on liver function, the following directly contribute to heightened inflammation with HCV:

1. Alcohol – There are many reasons why eliminating alcohol is imperative for living long with Hepatitis C. Researchers have demonstrated that alcohol promotes proliferation of Hepatitis C in human liver cells. Researchers at the Children’s Hospital in Philadelphia found that alcohol increases the activity of a protein called nuclear factor kappa B, which causes HCV to replicate. Aside from the cycle of inflammation that occurs with Hepatitis C, alcohol consumption on its own increases cytokine levels. Additionally, metabolized alcohol is believed to activate stellate cells directly. All of the chemical processes that occur when a person drinks alcohol exponentially worsen the damage that HCV does to the liver.

2. Marijuana – According to a study published in Clinical Gastroenterology and Hepatology, patients with HCV should not use marijuana (cannabis) daily. The researchers led by Terrault found that HCV patients who used cannabis daily were at significantly higher risk of moderate to severe liver fibrosis, or tissue scarring. Additionally, patients with moderate to heavy alcohol use combined with regular cannabis use experienced an even greater risk of liver fibrosis.

3. Fatty Food – Despite campaigns claiming that eliminating saturated fat from the diet preserves heart health, hepatologists agree that it also preserves liver health. Research from 2007 demonstrated that a high fat diet kills regulatory T cells in the liver. Less of these specialized immune cells allow a fatty liver to worsen to steatohepatitis, fatty liver with inflammation. This likely occurs because regulatory T cell death is associated with increased inflammatory cytokine production.

Although removing these three vices from one’s life may be a monumental life change for someone, it can also save their liver. The increase in inflammation that drinking alcohol, smoking marijuana and eating saturated fat can cause allows a liver with HCV to spiral into advanced liver disease. By abandoning these three unhealthful habits, the liver gets a respite from the inflammation cycle – perhaps enough for the body to break down some of the collagen matrix that contributes to the continuation of liver cell death.


References:

http://familydoctor.org, Hepatitis C, American Academy of Family Physicians, 2008.

http://pubs.niaaa.nih.gov, Alcohol and the Liver, National Institute for Alcohol Abuse and Alcoholism, 2008.

www.cdc.gov, Hepatitis C Fact Sheet, US Department of Health and Human Services, 2008.

www.hepctrust.org.uk, Liver damage and fibrosis during the chronic stage, Hepatitis C Trust, 2008.

www.medicalnewstoday.com, How Alcohol Use May Worsen Hepatitis C Infection, John Ascenzi, MediLexicon International, Ltd., 2007.

www.medicalnewstoday.com, Risk Of Hepatitis C-Related Liver Damage Increased By Regular Marijuana Use, MediLexicon International Ltd., 2008.

www.sciencedaily.com, High-fat Diet Makes Mice Susceptible To Liver Injury, ScienceDaily LLC 2008.

www.who.int, Hepatitis C, World Health Organization, 2008.

Posted by Editors at 02:00 PM --- Printer-friendly version

March 13, 2008

New, Interactive Internet Program Answers Hepatitis C Questions

A national, non-profit, public charity, the Caring Ambassadors Hepatitis C Program makes it easier for people with Hepatitis C to learn about the virus, its implications and up-to-date treatments. By utilizing the internet, people can get a customized response to their specific Hepatitis C concerns.

Caring Ambassadors Hepatitis C Program Launches Internet-Based Hep C Discussion PointTM to Assist People Living with Hepatitis C

www.marketwire.com

Mar 11, 2008

VANCOUVER, WA--(Marketwire - March 11, 2008) - The Caring Ambassadors Hepatitis C Program (CAP Hepatitis C), a national nonprofit organization, announces the Internet release of its new interactive medical management tool, Hep C Discussion Point™ at www.HepCChallenge.org. Hep C Discussion Point™ takes the user through a guided series of questions about their hepatitis C experience. Custom-built software analyzes the user's responses and generates a report with information and topics specific to the user's inputs. The report is designed to be used as both a learning tool for the patient and as a guide to facilitate communication and enhance the health care partnership between people living with hepatitis C and their doctors.

Hep C Discussion Point™ was developed by CAP Hepatitis C in conjunction with leading experts in the field of hepatology to help facilitate, inform, and enhance the therapeutic decision-making process by providing discussion points on state-of-the-art hepatitis C management. Hep C Discussion Point™ is a groundbreaking effort, and is the only tool of its kind designed exclusively for hepatitis C clients and their health care providers.

According to Lorren Sandt, the Hepatitis C Program Director, many of the calls CAP Hepatitis C receives are from patients who are confused by conflicting information they read or hear about regarding hepatitis C management. "When we designed Hep C Discussion Point™, we tried to address the most common and relevant questions we receive from those living with hepatitis C."

"We are excited about the launch of Hep C Discussion Point™ and are very pleased to offer this tool to the hepatitis C community, which includes both patients and their health care providers," said Dr. Tina St. John, Executive Director and Medical Director of the Caring Ambassadors Program. "Hep C Discussion Point™ represents a new and innovative approach to CAP Hepatitis C's commitment to ensuring that all persons living with hepatitis C have accurate and adequate information by which to make decisions that match their personal medical circumstances and health care goals."

Hepatitis C is the most common chronic, blood-borne viral infection in the United States. An estimated 5 million Americans are infected with the hepatitis C virus (HCV), the most common cause of chronic liver disease and adult liver transplantation in the U.S. No vaccine is available to prevent chronic hepatitis C but medications are available to clear the hepatitis C virus from the body in up to 50% of people treated.

About Caring Ambassadors Hepatitis C Program

The Caring Ambassadors Program is a 501(c)(3) nonprofit public charity dedicated to helping people with chronic and/or life-threatening diseases through information/education, awareness, public advocacy, and support. Founded in 2001, the organization is headquartered in Vancouver, Washington, U.S.A.

The Caring Ambassadors Hepatitis C Program (CAP Hepatitis C) is devoted exclusively to meeting the needs of the hepatitis C community. The CAP Hepatitis C mission is to improve the lives of people living with hepatitis C through information and awareness.

For additional information about the Caring Ambassadors Hepatitis C Program and Hep C Discussion Point™, contact Lorren Sandt at 360.816.4186 or Lorren@HepCChallenge.org.

Posted by Editors at 03:44 PM --- Printer-friendly version

March 07, 2008

Fast Food Likely to Accelerate Hepatitis C

According to new research from Sweden, grabbing a quick burger and fries is just as harmful to the liver as drinking alcohol. Since liver damage carries a graver risk to those living with Hepatitis C, eating low-fat meals is more important to this population than ever.

by Nicole Cutler, L.Ac.

New research from Sweden confirms that eating fast food can cause substantial liver damage. Luckily, the liver is one of our few organs capable of regenerating new, healthy cells. Thus, the injury inflicted from occasionally ordering dinner from a drive-through window can be reversed with a focused effort. Unfortunately, those battling Hepatitis C have a diminished ability to regenerate new liver cells and therefore have a harder time reversing liver damage. This means that the liver cell death incurred from eating the high-fat meals typical of fast food restaurants may be more detrimental to those already living with a chronic liver disease such as Hepatitis C.

The Hepatitis C virus (HCV) is one of the most common causes of chronic liver disease in the United States. Accounting for about 15 percent of acute viral hepatitis, 60 to 70 percent of chronic hepatitis and up to 50 percent of cirrhosis, end-stage liver disease and liver cancer, experts estimate that 4.1 million Americans are currently infected with HCV. While many more people live with HCV than die from it, the distinguishing factor between the two is learning how to prevent incurring further liver damage.

The HCV Disadvantage
The progression of liver disease is marked by a greater proportion of liver cell death than the creation of new, healthy liver cells. Due to a buildup of localized scar tissue, the following cascade of events demonstrates why those with HCV are prone to a worsening of their condition:

1. The Hepatitis C virus infects and kills liver cells.

2. In response to liver cell death, the body initiates an immune response, which causes inflammation.

3. In an attempt to limit the spread of infection, the immune system lays down collagen fibers between liver cells. These fibers are the building blocks of scar tissue.

4. Normally, as an infection or injury resolves, the collagen matrix enclosing the injury is dissolved and allows the tissue to return to normal. However, this web of collagen in people with chronic HCV grows more rapidly than it can dissolve.

5. As more collagen accumulates, the resulting scar tissue restricts living liver cells from access to nutrients and oxygen-rich blood.

6. The restricted access to blood causes even more quarantined liver cells to die. As such, HCV progressively scars the liver.

Since people living with HCV have this consistent battle being waged in their liver, they are more vulnerable to toxins that could damage this organ.

The Danger of Fast Food
Many things we breathe, ingest, swallow or otherwise absorb into our bodies pose a threat to the liver. A universally acknowledged cause of chronic liver disease, drinking alcohol is one of the most obvious ways to poison a liver. While most people might not categorize their lunch as toxic, millions of Americans are damaging their liver by eating fast food. A small Swedish study published in the February 2008 edition of the journal Gut found that even a brief fast-food binge combined with too little exercise can induce liver damage.

In this study, 18 slim, healthy participants stuck to a fast food diet, mostly consuming of hamburger meals from popular chains twice a day for four weeks while refraining from exercise. At the end of the experiment, participants not only gained an average of 16 pounds, but blood tests also showed evidence that those eating fast food incurred liver damage.

In the Swedish study, those gorging on fast food meals showed a dramatic increase in alanine aminotransferases (ALT) levels over a very short period of time. An enzyme produced in liver cells, ALT leaks into the bloodstream as liver cells are damaged. A result of liver cell damage, ALT elevations can be caused by any liver disease, hepatic inflammation or toxin.

Previous research has shown that a diet high in fat and calories, the hallmark of greasy, fast food, puts people at greater risk for obesity and type 2 diabetes, cardiovascular disease and heart failure. However, this Swedish study shows that doubling caloric intake without adding exercise also puts hepatic health in jeopardy. When excessive calories and fat overloads the liver’s ability to filter the blood, fat builds up in the liver cells and leads to liver damage. Considering that a majority of the study participants developed pathological ALT levels within just one week after eating fast food, this negative impact can occur relatively quickly.

Most people with HCV are aware that drinking alcohol causes liver cell death and can accelerate the severity of their liver disease. However, now their physicians must relay that alcohol is not the only thing they must navigate away from. Since eating a large quantity of high-fat foods (even for a short time period) while refraining from physical activity can damage the liver, people with HCV must avoid this combination. Since harboring the Hepatitis C virus makes recovery from liver damage a great challenge, hepatologists across the globe will be putting greater emphasis on sticking to a healthy, low-fat diet and adhering to an active lifestyle.


References:

http://abcnews.go.com, Fast Food: The Fast Track to Organ Damage, Radha Chitale, ABC News Internet Ventures, February 2008.

http://digestive.niddk.nih.gov, Chronic Hepatitis C: Chronic Disease Management, National Digestive Clearinghouse, 2008.

Diseases International http://en.wikipedia.org, Super Size Me, Wikimedia Foundatin Inc., 2008.

Kechagias, S, et al., Fast food based hyper-alimentation can induce rapid and profound elevation of serum alanine aminotransferase in healthy subjects, Gut, February 2008.

www.medpagetoday.com, Fast-Food Bender Can Convert to Liver Damage Swiftly, MedPage Today LLC, 2008.

Posted by Editors at 04:54 PM --- Printer-friendly version

Non-Invasive Testing for HCV Fibrosis Progression Improved

Michigan researchers have found that a new combination of markers may best predict the progression of cirrhosis for people with chronic Hepatitis C. Although requiring additional study for confirmation, these three serum fibrosis markers demonstrated a higher level of accuracy than other non-invasive tests.

Biomarker Panel May Improve Disease Staging in Chronic HCV

By Michael Smith, North American Correspondent, MedPage Today
Published: March 05, 2008
Reviewed by Robert Jasmer, MD; Associate Clinical Professor of Medicine, University of California, San Francisco

www.medpagetoday.com

ANN ARBOR, Mich., March 5 -- A panel of three biomarkers may be useful in identifying patients with chronic hepatitis C who have progressed to cirrhosis, researchers here found.

The three-marker panel was highly correlated to Ishak score in a prospective sub-study of the Hepatitis C Antiviral Long-term Treatment against Cirrhosis (HALT-C) Trial, according to Robert Fontana, M.D., of the University of Michigan, and colleagues.

The biomarkers were also more accurate than other non-invasive tests that have been proposed, the researchers reported in the March issue of Hepatology.

The three markers evaluated by the researchers were serum tissue inhibitor of matrix metalloproteinase-1 (TIMP-1), hyaluronic acid (HA), and platelet count.

All were significantly associated with cirrhosis in a univariate analysis (as were several other markers) but in multivariate analysis, the three biomarkers had an area under the receiver operating curve of 0.81.

(A receiver operating curve measures the trade-off between sensitivity and specificity for a given test; if the area under the curve were 1.0, the test would correctly identify both positives and negatives.)

By comparison, Dr. Fontana and colleagues said, three other published models -- the Lok model, the AST-to-platelet ratio index, and the cirrhosis discriminant score -- had lower areas under the curve of 0.79. 0.73, and 0.70 respectively.

For this study, the researchers compared the model's predictions with the biopsy results of 513 patients with chronic hepatitis C and Ishak scores of between two and six. Those with Ishak scores of five and six (38%) were considered to have cirrhosis, while the remaining 62% had fibrosis.

Among those patients, the researchers found, the model would have correctly categorized 153 patients as having a low likelihood of cirrhosis with 86% accuracy.

An additional 146 subjects would have been categorized as having a high likelihood of cirrhosis with 73% accuracy.

The study was limited by the nature of the HALT-C patient population, the researchers said, which meant that there was no independent cohort of patients who also had stored serum available for testing for comparison.

Also, they noted, the model was not tested in an external validation cohort.

Nevertheless, the model "distinguished patients with non-cirrhotic (chronic hepatitis C) from those with cirrhosis" and "performed significantly better than other models based on routine laboratory tests," the researchers concluded.

The implication is that serum fibrosis markers "provide useful, incremental information in estimating disease stage" in chronic hepatitis C that can be obtained without biopsy, they said.

The study was supported by the National Institute of Allergy and Infectious Diseases, the National Cancer Institute, the National Center for Minority Health and Health Disparities, the National Center for Research Resources of the NIH, and Hoffmann-La Roche Inc.

Dr. Fontana reported being on the speakers bureau for Hoffmann-La Roche.

Posted by Editors at 02:52 PM --- Printer-friendly version

March 03, 2008

Re-Used Equipment at Nevada Clinic May Have Spread Hepatitis C

People treated at a Nevada Health Center may have been infected with Hepatitis C from an unsafe medical procedure. Thousands of people are being notified that they may have been exposed to this and other blood-borne viruses from the re-use of contaminated anesthesia equipment.

Nevada Hepatitis C Outbreak Tied to Las Vegas Clinic. Thousands Now At Risk for Hepatitis, HIV

Date Published: Thursday, February 28th, 2008

www.newsinferno.com

Hepatitis C and other blood borne diseases now threaten thousands of people in Nevada, thanks to the unsafe way anesthesia was administered at the Endoscopy Center of Southern Nevada in Las Vegas. At least six people who received treatment at the Endoscopy Center of Southern Nevada have already tested positive for Hepatitis C, but health officials in the state have urged another 40,000 to be tested for the virus, as well as HIV.

Hepatitis C is a blood disorder that is transmitted through blood-to-blood contact. Hepatitis C for the most part is asymptomatic and often leads to chronic, and long-term infection resulting in approximately 70% of those infected developing liver disease. Hepatitis C is a risk factor for liver cancer and can lead to the need for a liver transplant. HIV is the virus that causes AIDS, and is transmitted through the exchange of bodily fluids, including blood-to-blood contact.

The Endoscopy Center of Southern Nevada Health has been under investigation since early January, after health officials learned of three people who had been diagnosed with Hepatitis C. According to the Southern Nevada Health District, a total of six people contracted Hepatitis C after being treated at the Endoscopy Center of Southern Nevada. Five of them were treated the same day in late September; the sixth is believed to have been infected in July, the district said. The Southern Nevada Health District investigation revealed that “unsafe injection practices related to the administration of anesthesia medication might have exposed patients to the blood of other patients,” the statement said.

The Hepatitis C virus may have been spread when clinic staff reused syringes and used a single dose of anesthesia medication on multiple patients, the district said. A syringe would become contaminated by the backflow of blood when patients with a blood-borne disease were injected with medication, health officials said. That syringe, in turn, would be reused to withdraw medication from a different vial. That vial could become contaminated and result in infection.

The Southern Nevada Health District said that the unsafe practices had been in place for several years at the Endoscopy Center of Southern Nevada, and may have put others at risk. About 40,000 patients who received injections of anesthesia at the clinic will be told of the potential exposure in letters arriving next week. Anyone who received anesthesia at the clinic from March 2004 to Jan. 11 should be tested for the virus, along with Hepatitis B and HIV. The Southern Nevada Health Districts patient notification will be the largest of its kind in the country.

This is not the first time an outbreak of Hepatitis was blamed on medical practitioners who reused syringes or reused multidose vials of anesthesia on more than one patient. Late last year, the New York State Department of Health warned thousands of people treated by Long Island anesthesiologist Harvey Finkelstein that they were at risk for Hepatitis C, B and HIV. Finkelstein also was known to reuse syringes. At least one person is known to have contracted Hepatitis C as a result of Finkelstein’s unsanitary practices, and another six patients tested positive for the disease, although it is not absolutely certain that the virus was the result of Finkelstein’s treatment. Another six tested positive for Hepatitis B.

Posted by Editors at 09:22 AM --- Printer-friendly version

February 28, 2008

Update: What You Need to Know About Grapefruit and Hepatitis C

Even though there are more reasons than ever for those with chronic Hepatitis C to have a daily glass of grapefruit juice, there is a catch. Unfortunately, there is a long list of medications that can be dangerous when combined with this fruit.

by Nicole Cutler, L.Ac.

In the aftermath of recent research from Massachusetts, those with Hepatitis C are likely consuming more grapefruit than ever. By concluding that a compound in grapefruit likely prevents the Hepatitis C virus (HCV) from proliferating, this citrus fruit may accompany antiviral medication prescriptions in the future. However, anyone with a chronic illness considering adding grapefruit or its juice to their shopping list must be aware of the danger it could potentially inflict.

The Research
An investigative team from the Massachusetts General Hospital Center for Engineering in Medicine recently announced details about HCV propagation and the role grapefruit may have in hindering it. Apparently, Hepatitis C virus binds to a very low-density lipoprotein (vLDL) before it is secreted from liver cells to re-infect additional liver cells. Otherwise referred to as the bad cholesterol, vLDL functions as the body’s internal transport mechanism for lipids. According to lead author Yaakov Nahmias, Ph.D., “By finding that HCV is secreted from infected cells by latching onto vLDL, we have identified a key pathway in the viral lifecycle.”

Since HCV does not integrate its genetic material into the DNA of infected cells the way HIV does, totally clearing the virus is possible if new cells were not being infected. Therefore, interfering with the transport of HCV out of cells holds a great deal of therapeutic promise.

Scientists found that by blocking vLDL with a compound in grapefruit, Hepatitis C lost its vehicle and thus was stopped from expansion. Grapefruit's bitter taste is caused by the presence of the flavonoid naringin, which is metabolized into naringenin, an antioxidant known to reduce the secretion of vLDL from liver cells. The Massachusetts investigators confirmed that naringenin reduces the secretion of Hepatitis C from infected cells.

Another Grapefruit Advantage
Although focusing on a different component of the grapefruit, previous research supports the hepatic potential of grapefruit. Phytonutrients in grapefruit, called limonoids, inhibit tumor formation by promoting the formation of glutathione-S-transferase, a detoxifying enzyme. This enzyme sparks a reaction in the liver that helps to make toxic compounds more water soluble for excretion from the body.

Since those with chronic Hepatitis C are at a greater risk for developing cancer of the liver, inhibiting tumor formation is especially important. By helping the liver clear out cancer-causing toxins, limonoids’ promotion of detoxification enzymes is a simple way to ward off the development of cancer.

Grapefruit Warning
While just a few of grapefruit’s many health benefits are described above, there is a bittersweet side to this popular fruit. Grapefruit and its juice can be dangerous to people who take certain medications.

Amy Karch, RN, MS, of the School of Nursing at the University of Rochester Medical Center, an expert on drug interactions, explains that grapefruit juice is one of the foods most likely to cause problems with medications. The cytochrome P-450 3A4 enzyme breaks down grapefruit juice into useful components for the body, just like it breaks down dozens of medications. Grapefruit juice can block this enzyme, making it easier for medications metabolized by the same pathway to pass rapidly from the digestive system to the bloodstream. The result is blood levels of the drug rising faster and higher than normal. In some cases the abnormally high medication levels can be dangerous.

Consisting of more than 50 medications, interactions with grapefruit juice are well-known among researchers and clearly documented on warning labels. However, people commonly fail to comprehensively read the warning labels about drug-food interactions. In addition, it doesn’t take much grapefruit juice to boost the levels of drugs that are susceptible. A single glass can produce a 47 percent reduction of the intestinal enzyme that regulates absorption. Since the effect of grapefruit juice wears off slowly, a third of its impact is still evident after 24 hours.

While this list is not complete, some of the drugs interacting with grapefruit include:

· Anxiety: Xanax, Buspar, Versed, Halcion
· Depression: Luvox, Zoloft
· Allergies: Allegra
· Abnormal heart rhythm: Cordarone, quinidine
· Heart disease/stroke/blood clots: Coumadin
· Epilepsy: Tegretol
· Cancer: Cyclophosphamide, etoposide, ifosfamide,
tamoxifen, vinblastine, vincristine
· Cough: Dextromethorphan (found in many over-the-counter cold
medicines)
· HIV: Agenerase, Crixivan, Viracept, Norvir, Fortovase
· Prostate enlargement: Proscar
· Heart disease/High blood pressure: Coreg, Cardizem, Plendil, Cardene, Adalat, Procardia, Nimotop, Sular, Covera, Calan, Verelan
· Erectile dysfunction: Viagra, Cialis
· Asthma/Emphysema: Theophylline
· High cholesterol: Lipitor, Lescol, Mevacor, Zocor
· Pain: Alfenta, Duragesic, Actiq, Sufenta
· Infection: Biaxin, Sporanox, erythromycin, troleandomycin

As a person living with a chronic disease, those with HCV may have additional health issues warranting the use of medication. If unsure of a medication’s food interaction data, always contact a pharmacist to be sure.

The new evidence supporting the use of grapefruit in the fight against HCV is exciting, both because it may lead to viral elimination and because it can be done with an all-natural, well-known food source. However, before ordering your next case of grapefruit, make certain you don’t put yourself in jeopardy. If indulging in this sweet, tart member of the citrus family, be certain that any medications you take will not interact with your grapefruit.


References:

http://en.wikipedia.org, Very low density lipoprotein, Wikimedia Foundation, Inc., 2008.

www.health.harvard.edu, Grapefruit and medication: A cautionary note, President & Fellows of Harvard College, 2008.

www.peacehealth.org, Grapefruit Juice and Medication, Healthwise, 2008.

www.sciencedaily.com, Grapefruit Compound May Help Combat Hepatitis C Infection, ScienceDaily LLC, 2008.

www.sciencedaily.com, Grapefruit Juice And Medication Can Be A Dangerous Mix, ScienceDaily LLC, 2008.

www.whfoods.com, Grapefruit, The George Mateljan Foundation, 2008.

Posted by Editors at 09:44 AM --- Printer-friendly version

February 26, 2008

Agreement to Bring Rapid, Oral HCV Test Outside the U.S.

Two big pharmaceutical companies, Schering-Plough and OraSure, have agreed to work together to deliver a rapid, oral Hepatitis C test. In addition to their collaboration within the United States, this new agreement focuses on bringing this technology to additional markets.

AFX News Limited

Schering-Plough, OraSure Tech collaborate on oral hepatitis C test outside U.S.

www.forbes.com

02.11.08, 9:41 AM ET

NEW YORK (Thomson Financial) - Schering-Plough Corp. and OraSure Technologies Inc. Monday agreed to collaborate on the development and promotion of an oral hepatitis C virus (HCV) test outside the U.S.

The test will use OraSure's OraQuick technology platform.

Under the agreement Schering-Plough (nyse: SGP - news - people ) will reimburse OraSure for certain development costs and will provide payments to OraSure based on the achievement of certain regulatory and commercial milestones in international markets. Schering-Plough will provide promotional support while OraSure will make all sales and retain the rights to market and sell the test in all markets throughout the world.

The agreement builds upon the existing collaboration announced in January 2007 to develop and promote a rapid oral HCV test in the U.S. physicians' office market.

'We believe the global market opportunity for a rapid HCV test is significant and this expansion of our collaboration with Schering-Plough should help drive the adoption of this important product around the world,' OraSure said.

OraSure is a Bethlehem, Pa.-based company that specializes in oral fluid specimen collection devices and diagnostic products. Its stock closed Friday at $7.61.

Shares of Schering-Plough, a Kenilworth, N.J.-based advanced drug therapy company, closed Friday at $19.77.

Melinda Peer

mp/pc

Copyright Thomson Financial News Limited 2007. All rights reserved.

Posted by Editors at 09:55 AM --- Printer-friendly version

February 18, 2008

How Grapefruit Reduces the Spread of HCV

Massachusetts researchers have found that a compound in grapefruit blocks the Hepatitis C virus from infecting additional cells. Since the Hepatitis C virus depends on cholesterol metabolism to proliferate, future trials may prove that lipid-lowering drugs or supplements, such as that found in grapefruit, may inhibit the virus.

Grapefruit compound may help fight hepatitis C infection

www.tamilstar.com

Feb 14, 2008, 05:21

A compound naturally occurring in grapefruit and other citrus fruits may help get rid of hepatitis C virus, according to a study published in an upcoming issue of the journal Hematology.

The study led by researchers at the Massachusetts General Hospital Center for Engineering in Medicine showed naringerin, a flavonoid found in grapefruit, blocks the secretion of hepatitis C virus from infected cells.

Hepatitis C virus needs to latch onto a very low-density lipoprotein (vLDL, a so-called bad cholesterol) to pass on the infection to other cells in the liver.

The effect was observed in cultured cell lines.

"These results suggest that lipid-lowering drugs, as well as supplements, such as naringenin, may be combined with traditional antiviral therapies to reduce or even eliminate HCV from infected patients," said Yaakov Nahmias, PhD, of the MGH-CEM, the paper's lead author.

"Identifying the route by which HCV is released from cells introduces a new therapeutic target," said Martin Yarmush, MD, PhD, director of the MGH-CEM and the paper's senior author.

"That pathway's dependence on cholesterol metabolism could allow us to interfere with viral propagation to other cells and tissues, using tools already developed for atherosclerosis treatment."

Early studies showed hepatitis C virus needs vLDL to maintain its infection and naringenin can reduce secretion of vLDL from liver cells. The current study was meant to examine whether the compound might also lower HCV secretion from infected cells.

Hepatitis C virus is the leading cause of chronic viral liver disease in the United States that infects about 3 percent of the world population.

Eighty percent of persons with hepatitis C have no signs or symptoms but common symptoms include jaundice, fatigue, dark urine, abdominal pain, loss of appetite and nausea. Often it transmits from one person to another through contact of blood.

The current antiviral medications can treat 50 percent of cases, but 70 percent are expected to develop chronic infection, leading to cirrhosis or liver cancer in the end.

The researcher said naringerin or other lipid-lowering drugs could be used with other antiviral medications to treat hepatitis C infection if the effect is confirmed in human trials.

Posted by Editors at 01:45 PM --- Printer-friendly version

January 28, 2008

Hep C May Affect More than the Liver

A new study from Bulgaria proves that Hepatitis C infection is not only a liver disease, but affects many different parts of the body. Their research demonstrates that over three quarters of people with Hepatitis C have extra-hepatic manifestations and it helps physicians identify who is at the greatest risk.

Hepatitis C Virus Affects Many Organs And Tissues, Not Just Liver

www.sciencedaily.com

ScienceDaily (Jan. 18, 2008) — In 1994, the team of Tchernev and Petrova from Alexandrovska Hospital in Sofia examined a female patient with liver cirrhosis caused by chronic Hepatitis C virus (HCV). They were intrigued by the patient's many extra-hepatic manifestations -- vascular lesions on the lower limbs, acute pain in the joints, intense tingling of the fingers, and extreme labor-impairing fatigue. They were also intrigued by the presence of cryoglobulins in the patient's blood. Two years later, the patient developed enlarged lymph nodes on the neck. When one of the nodes was histologically tested, the patient was found to have lymphoma.

This case spurred the interest of the investigators in the extra-hepatic manifestations and complications of HCV infection, and for over a decade they studied the links between HCV infection, cryoglobulinemia, and lymphoma.

Hepatitis C virus is a major health problem worldwide, and more than 3 percent of the world's population is infected with HCV. Despite popular belief, HCV is not only a liver disease, but affects many organs, tissues, and systems.

In a new study of 136 Bulgarian patients with HCV, the team of Tchernev and Petrova found 76.5% of the patients had extra-hepatic manifestations. Common manifestations were fatigue (59.6%), renal impairment (25%), type 2 diabetes (22.8%), paresthesia (19.9%), arthralgia (18.4%), and purpura predominantly of the lower limbs (17.6%). Over 37% of the patients had cryoglobulins, and 8.8% had B-cell lymphoma.

The study found positive links between the presence of extra-hepatic manifestations and age, female gender, duration of the infection, infection by transfusion of blood and blood products, and extensive liver fibrosis. Therefore, elderly women with chronic HCV and advanced liver fibrosis, who were infected by transfusion during childbirth, are at the highest risk of developing extra-hepatic manifestations of HCV infection.

The study also showed most extra-hepatic manifestations of HCV infection are associated with the presence of cryoglobulins. In particular, the risks of developing B-cell non-Hodgkin lymphoma are much higher in cryoglobulin-positive than in cryoglobulin-negative patients. In the study, 17.6% of cryoglobulin-positive patients had lymphoma, whereas only 3.5% of cryoglobulin-negative patients did.

Given the prevalence of HCV around the world, it is important for physicians to recognize the extra-hepatic signs and symptoms of HCV infection. Patients who exhibit such manifestations should be tested for HCV infection. This can lead to prompt diagnosis and effective treatment of the infection before the development of cryoglobulinemia, when treatment gives poor results or is ineffective.

Journal reference: Stefanova-Petrova DV, Tzvetanska AH, Naumova EJ, Mihailova AP, Hadjiev EA, Dikova RP, Vukov MI, Tchernev KG. Chronic hepatitis C virus infection: Prevalence of extrahepatic manifestations and association with cryoglobulinemia in Bulgarian patients. World J Gastroenterol 2007; 13(48): 6518-6528 http://www.wjgnet.com/1007-9327/13/6518.asp

Adapted from materials provided by World Journal of Gastroenterology, via EurekAlert!, a service of AAAS.

Posted by Editors at 11:41 AM --- Printer-friendly version

Roche Stands By Their HCV Drugs Citing "IDEAL" Study Design Issues

Indicating patient preference for PegIntron™, Schering-Plough recently publicized their Hepatitis C trial, "IDEAL." However, competitor Roche indicates several IDEAL trial design issues that make for a poor comparison to their PEGASYS® with COPEGUS®.

Media Release

Contacts:
Brad Jenkins, Roche
+41 61 68 86404

Michelle Marchione
Axon Communications
+1 416 848 1419

Roche responds to announcement of “IDEAL” hepatitis C study

BASEL – January 14, 2008 – Following an announcement from Schering-Plough, Roche today affirmed the value of PEGASYS® (peginterferon alfa-2a) in combination with COPEGUS® (Roche’s brand of ribavirin) as the market-leading treatment for patients with hepatitis C. Despite clear biases in the design of the “IDEAL” study that potentially favoured patients taking PegIntron™ (peginterferon alfa-2b) regimens – particularly the ribavirin dose reduction protocol – the study results have shown that patients treated with a PEGASYS regimen had a similar chance of being successfully treated for hepatitis C.

“I do not expect that the results of the IDEAL study will meaningfully impact clinical practice, except to inform physicians on the appropriate dosing of PegIntron and to reinforce the already widely-accepted view that optimising ribavirin dosing throughout treatment is critical to achieving success and preventing treatment relapse in hepatitis C,” said Douglas Dieterich, M.D., Professor of Medicine in the Division of Hepatology at Mt. Sinai School of Medicine in New York, New York.

In 2001, the U.S. Food and Drug Administration (FDA) required Schering-Plough to conduct a post-approval commitment trial to determine if a lower dose of PegIntron (1.0 mcg/kg) was as effective as the approved dose of 1.5 mcg/kg, both in combination with identical ribavirin regimens.1 A third arm was added to the study in which patients received PEGASYS 180 mcg with a different ribavirin dosing schedule. This mismatch of ribavirin dosing introduces several potential biases into the study because experts agree that an optimised dose of ribavirin, with either pegylated interferon, is critical to achieving success in hepatitis C treatment. In particular, maintaining a full dose of ribavirin has shown an important ability to reduce relapse following the end of treatment.

“PEGASYS quickly became the market leader after its launch, based on robust clinical data and patient and physician preference. We are convinced that physicians and patients will continue to choose the PEGASYS plus COPEGUS combination therapy based on positive experience and sound clinical evidence,” said Rob Mitchell, Head of Viral Diseases Strategic Marketing at Roche. “Our current focus at Roche is on advancing the treatment of hepatitis C by optimising doses and duration of PEGASYS and ribavirin in patients with unmet medical need, while developing new compounds that have the potential to offer a successful outcome to even more patients.”

Roche believes that it is critical for patients and physicians to receive complete information to fully understand the results of “IDEAL” so that treatment decisions can be based on scientific data.
###

Please see below for additional information about the “IDEAL” trial, Roche and PEGASYS including important safety information.

“IDEAL” Trial Design Issues
· Starting doses of ribavirin were different in the PegIntron and PEGASYS arms of the study
· The design calls for a more drastic ribavirin dose reduction for side effect management in most patients in the PEGASYS arm compared to patients in the PegIntron arms; in some cases, ribavirin dose reductions for patients in the PEGASYS arm were three times greater than for patients in the PegIntron arms. This is important because a substantial number of patients being treated for hepatitis C require their ribavirin dose to be reduced to manage side effects, and this could have an impact on the efficacy of the regimen
· The PEGASYS arm was not blinded, meaning that patients and physicians knew which treatment was being administered. Many comparative studies are blinded to ensure that bias does not compromise the results
· Erythropoetin (EPO) is a medication that is often given to treat ribavirin-related anemia and help patients maintain a higher ribavirin dose. However, physicians could only prescribe EPO after the first dose ribavirin reduction in the “IDEAL” trial. Since patients in the PegIntron arms generally had smaller ribavirin dose reductions, this introduces another potential bias and means those PegIntron patients were potentially able to maintain a higher dose of ribavirin compared to PEGASYS patients

Efficacy of PEGASYS plus COPEGUS Combination Therapy
PEGASYS was launched by Roche in 2002 and quickly became the leading treatment for patients with hepatitis C. PEGASYS plus COPEGUS is the only pegylated interferon combination regimen to have demonstrated significantly superior benefits over conventional interferon combination therapy across all HCV genotypes, irrespective of viral load.2-4 The combination of PEGASYS and COPEGUS consistently shows high cure rates – up to 66% overall sustained virological response – across a number of large, randomised clinical studies including in patients with difficult-to-cure disease such as genotype 1 HCV, cirrhosis, and HIV-HCV co-infection.2-7

About Roche
Headquartered in Basel, Switzerland, Roche is one of the world’s leading research-focused healthcare groups in the fields of pharmaceuticals and diagnostics. As the world’s biggest biotech company and an innovator of products and services for the early detection, prevention, diagnosis and treatment of diseases, the Group contributes on a broad range of fronts to improving people’s health and quality of life. Roche is the world leader in diagnostics and drugs for cancer and transplantation, a market leader in virology and active in other major therapeutic areas such as autoimmune diseases, inflammation, metabolism and central nervous system. In 2006 sales by the Pharmaceuticals Division totalled 33.3 billion Swiss francs, and the Diagnostics Division posted sales of 8.7 billion Swiss francs. Roche employs roughly 75,000 worldwide and has R&D agreements and strategic alliances with numerous partners, including majority ownership interests in Genentech and Chugai.

###

All trademarks used or mentioned in this release are protected by law.

References:

1. FDA letter to Schering-Plough, August 7, 2001. Accessed Nov. 26, 2007 at: http://www.fda.gov/cder/foi/appletter/2001/pegsche080701L.htm
2. Swan, T. Expediency, Cost-Cutting, Expediency Trump Science in Clinical Development Plan for Peg-Intron: The head-to-head that wasn’t. TAGLine 2003: 10(10)1-4. Also available at: http://www.aidsinfonyc.org/tag/taglines/0312.pdf
3. Raymond, D. The Real IDEAL: Peg-Intron vs. Pegasys. Hepatitis C Harm Reduction Project Web site. Accessed Dec. 17, 2007 at: http://hepcproject.typepad.com/hep_c_project/2004/05/the_real_ideal_.html
4. Hadziyannis SJ, Sette H, Jr., Morgan TR, et al. Peginterferon-alpha2a and ribavirin combination therapy in chronic hepatitis C: a randomized study of treatment duration and ribavirin dose. Ann Intern Med 2004;140(5):346-55.
5. Torriani FJ, Rodriguez-Torres M, Rockstroh JK, et al. Peginterferon Alfa-2a plus ribavirin for chronic hepatitis C virus infection in HIV-infected patients. N Engl J Med 2004;351(5):438-50.
6. Fried MW, Shiffman ML, Reddy KR, et al. Peginterferon alfa-2a plus ribavirin for chronic hepatitis C virus infection. N Engl J Med 2002;347(13):975-82.
7. Marcellin P, Brillanti S, Cheinquer H. Peginterferon alfa-2a (40KD) (PEGASYS) plus ribavirin (COPEGUS) is an efficacious and safe treatment for chronic hepatitis C (CHC) in patients with compensated cirrhosis. . In: 38th Annual Meeting of the European Association for the Study of the Liver (EASL) July 3-6; 2003; Geneva, Switzerland; 2003.

Posted by Editors at 11:40 AM --- Printer-friendly version

Non-Hodgkin's Lymphoma Less Likely with Hepatitis C Treatment

After following participants for one year, a large Japanese study concludes that treating Hepatitis C aggressively reduces the occurrence of non-Hodgkin's lymphoma.

Treatment of Hepatitis C Reduces Incidence of Non-Hodgkin’s Lymphoma

http://professional.cancerconsultants.com

Researchers from Japan have reported that viral elimination in hepatitis C virus (HCV) infected patients reduces the incidence of non-Hodgkin’s lymphoma (NHL). The details of this study appeared in the December, 2007 issue of the American Journal of Medicine.

Hepatitis C affects approximately 170 million individuals worldwide. Following acute infection, the virus persists in many patients and a minority of patients develop chronic disease. Chronic hepatitis can progress slowly over many decades to chronic active hepatitis and cirrhosis, ultimately leading to end-stage liver disease or hepatocellular carcinoma. Studies from other countries, but not from the United States, have also shown an increased incidence of NHL in patients with HCV infection. Some researches suspect that this correlation can only be observed in populations where HCV is highly prevalent.

The current study was carried in 501 patients with HCV infection who had never received interferon and 2,708 patients who had received interferon. These authors reported that by one year, 0.6% of untreated patients had developed NHL which by 5 years had increased to 2.3% and by 10 years to 2.6%. In contrast, there were no cases at 5, 10 and 15 years for the 1,048 patients with HCV infection who had sustained virologic response. The remaining patients who were treated and had persistent viral infection had an incidence of NHL of 0.4% at the fifth year, 1.5% at the tenth year and 2.6% at the fifteenth year.

Comments: These data support the concept that treating HCV infection aggressively can reduce the incidence of NHL.

Posted by Editors at 11:39 AM --- Printer-friendly version

HCV Alert: Another Doctor Reuses Syringes

Unfortunately, another Long Island doctor put patients in jeopardy of acquiring Hepatitis C when it was discovered that he reused syringes between patients. Unlike the Finkelstein case, New York authorities are moving quickly to inform the public and notify patients.

Another Long Island Doctor Commits Malpractice By Reusing Syringes

www.newsinferno.com

Date Published: Wednesday, January 16th, 2008

It’s been revealed that another Long Island doctor has reused syringes when administering injections to patients. Dr. E. Jacob Simhaee, a Manhasset-based obstetrician-gynecologist reused syringes when administering flu shots to at least 36 patients last fall. Simhaee is not the first Long Island doctor to commit such medical malpractice. Late last year, it was learned that Dix Hills doctor Harvey Finkelstein had infected several of his patients with blood borne diseases after reusing syringes.

According to the New York State Department of Health, Simhaee is contacting these patients in writing. The state Department of Health composed the letter for the physician. Simhaee was asked to sign the letter and is also contacting patients by telephone. The state initiated its investigation of Simhaee’s practice in December following a complaint filed with the Nassau County Department of Health.

The state’s release of information yesterday contrasts sharply with its handling of the Dr. Harvey Finkelstein case. It waited three years before telling the public last fall and notifying nearly 11,000 patients that Finkelstein had reused syringes—exposing thousands of patients to blood-borne pathogen infections—resulting in transmission of hepatitis C. As of Tuesday, 13 of Finkelstein patients have tested positive for hepatitis B and nine for hepatitis C. The state said it is impossible to determine whether Finkelstein’s office was the source of these infections. Finkelstein has more malpractice settlements than any other pain-management specialist on Long Island and, was sued, on average, once or twice yearly. Fifteen suits concerned epidural injections; at least 10 led to settlements. On his resume—posted on his now offline Web site—Finkelstein was described as a 1985 fellow in pediatric and cardiac anesthesia and a 1986 fellow in pain management via Stony Brook Hospital. A hospital spokeswoman said they were not accredited to offer fellowships in pain management until 1994, in pediatric anesthesia until recently, and are not accredited in cardiac anesthesia.

Mary Curtis, Nassau’s deputy executive of health and human services said, regarding the Simhaee investigation, “It’s amazing that in this amount of time, they conducted an investigation and made a notification,” adding, “The state and Nassau County did a great job. We’ve really learned from the past.” State senator Kemp Hannon (R-Garden City) feels the discovery of a second such case might warrant legislative action. “We’re going to have to look into the prohibition of multiple-use vials or limiting the use of syringes to single-use syringes,” he said.

As with Finkelstein, the department determined Simhaee used a single syringe, which held up to six doses, on multiple patients; infection-control procedures require a new syringe be used for each patient. The state said no diseases have been transmitted and Simhaee has cooperated fully. Simhaee’s patients who received the flu shot between September and December are being urged to be tested for hepatitis C, hepatitis B, and HIV and to be revaccinated against the flu.

According to Simhaee’s attorney, Craig Schaum of Garden City “This is a very highly respected doctor who has been cooperating in every way with state and county officials and will continue to do so.” Simhaee graduated in 1982 from the Albert Einstein School of Medicine at Yeshiva University in the Bronx, according to the state health department Web site; completed his graduate medical education at Maimonides Medical Center in the Bronx in obstetrics and gynecology; and is board certified in obstetrics and gynecology.

Posted by Editors at 11:35 AM --- Printer-friendly version

January 17, 2008

Fibrin Glue May Have Transmitted HCV

Used in Japan in the 1980s, a surgical adhesive made from fibrin is another suspect for the transmission of Hepatitis C. Primarily utilized for treating burns, nosebleeds and to aid in plastic surgery, fibrin glue may have been tainted with HCV-infected fibrinogen before proper testing was conducted.

Fibrin glue used for burns, facelifts

The Yomiuri Shimbun

www.yomiuri.co.jp

Fibrin glue, a surgical adhesive linked to the transmission of the hepatitis C virus, was used in a wide variety of areas, such as dealing with burns, stopping nosebleeds or in plastic surgery, according to doctors and other sources.

The tainted glue is estimated to have been used on about 79,000 patients, and those infected with the virus via the glue are eligible for relief under a law enacted Friday offering blanket relief to HCV suffers who contracted the disease through tainted blood products.

However, many patients do not realize the wide extent of the glue's use, causing delays in the investigation into how many patients have been infected with the virus via the glue. "The Health, Labor and Welfare Ministry should alert the public by announcing the names of hospitals that used the glue and for what kind of treatment the glue was often used," said Shiro Iino, former health ministry official in charge of research into issues concerning hepatitis.

The contaminated glue was made from a combination of substances including HCV-tainted fibrinogen manufactured by the defunct Green Cross Corp. mainly between 1981 and 1987. The glue was mostly used for stopping bleeding or as an adhesive during surgery. While it was not formally approved under the Pharmaceutical Affairs Law, Green Cross promoted the glue by issuing a booklet that gave details of how the glue could be used, resulting in many hospitals using it.

A private hospital in western Japan started using the glue from around 1982 as a surgical adhesive when it conducted major skin grafts on patients who had suffered severe burns. "As the glue had hemostatic and adhesive properties, we could conduct surgery quickly. I think we used the glue in treating between 10 to 20 patients a year," a doctor at the hospital said.

The hospital stopped using the glue in the late 1980s, when the risk of hepatitis infection came to light. They have not confirmed any cases of HCV infection via the glue.

"We no longer have the medical records of the patients as the storage term has already expired. But I think it's possible some patients will have contracted HCV via the glue as it was applied directly to the surface of the burned skin," the doctor said. "What has happened to those patients subsequently is a concern."

Meanwhile, a doctor in the ear, nose and throat department of another hospital in western Japan used the glue in the mid-1980s to treat patients suffering from nosebleeds. When it was difficult to identify from which part of the nose a patient was bleeding, the doctor filled the nose with the glue to stop the bleeding. The treatment was conducted on about 20 patients.

Several years ago, the doctor informed patients about the possibility of infection, and recommended them to undergo medical tests. But results obtained did not show any infection.

"As the glue was removed from the nose after treatment, that might have lowered the infection risk," the doctor said.

In the 1980s, the glue was also used in plastic surgery. According to an academic journal, an aesthetic plastic surgeon in Tokyo used the glue as a hemostatic agent or as a surgical adhesive in facelift operations.

According to a report submitted in 2001 to the Health, Labor and Welfare Ministry by the then Welfide Corp., which took over Green Cross, the glue was used in about 270 medical procedures, including some related to the treatment of pneumothorax and stomach ulcers.
(Jan. 15, 2008)

Posted by Editors at 03:15 PM --- Printer-friendly version

Proteomic Profiling Improves Liver Cancer Identification

Boston researchers have demonstrated that proteomics demonstrates a higher specificity and sensitivity for detecting liver cancer than traditional methods. This breakthrough may detect liver cancer tumors earlier, when they are easier to treat.

Proteomic profiling shown more accurate than traditional biomarkers in identifying liver cancer

Contact: Bonnie Prescott
bprescot@bidmc.harvard.edu
617-667-7306
Beth Israel Deaconess Medical Center

www.eurekalert.org

BOSTON – As the incidence of liver cancer continues to grow-- fueled in large part, by rising rates of hepatitis C infections – so too does the need for tests to help diagnose the disease at an earlier stage. A study appearing in the January 15 issue of Clinical Cancer Research demonstrates that a novel mass-spectrometry based form of proteomic profiling is more accurate than traditional biomarkers in distinguishing liver cancer patients from patients with hepatitis C liver cirrhosis, particularly with regard to identifying patients with small, curable tumors. Led by researchers at Beth Israel Deaconess Medical Center (BIDMC), the study could help lead to earlier diagnostic methods – and subsequent treatments -- for liver cancer.

“Proteomics represents a potentially powerful tool for the serologic recognition of protein profiles associated with cancer,” explains co-senior author Towia Libermann, PhD, Director of the Genomics Center at BIDMC and Associate Professor of Medicine at Harvard Medical School.

“Although this particular proteomics technology, SELDI-TOF MS [surface enhanced laser desorption/ionization time of flight mass spectrometry] had alrea